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1 Introduction Kidney disease is a major global non-communicable disease (NCD), with a global prevalence exceeding 10% [affecting ~850 million people when including acute kidney injury (AKI), kidney failure, and dialysis/transplant recipients] ( Cell death is a finely regulated process that occurs through various molecular pathways ( Recent studies have confirmed that the kidney is particularly susceptible to redox imbalance, and ferroptosis plays a significant role in the pathophysiology of various kidney diseases, emerging as a new research hotspot in the field of renal fibrosis ( Figure 1

Exposure to 17-estradiol upregulates the anti-apoptotic protein Bcl-2 while downregulating Bcl-XL, highlighting hormone-mediated modulation of apoptosis ( Figure 3 ) ( Figure 3 Human epidermal growth factor receptor 2/neuroglialoblastoma-derived oncogene (HER/2neu), a proto-oncogene located on chromosome 17q11.2-q12, is a well-recognised tumour-associated marker

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