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Early after liver resection, regeneration ability is insufficient due to the shortage of liver cells and liver volume, high pressure in the portal vein, long sustained time and small liver syndrome that is characterized by postoperative liver dysfunction, high blood bilirubin with prolonged cholestasis, blood coagulation disorders, portal hypertension, and the emergence of serious ascites

FAK integrates growth-factor and integrin signals to promote cell migration

Patients who stay on therapy longer see cumulative savings and better outcomes