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Together, these results suggest that enhanced alpha-cell GLP-1 production may be an early response to metabolic dysregulation, suggesting that the adaptation to metabolic stress involves a greater reliance on this intra-islet paracrine signaling for glucose homeostasis

A dosis teraputicas, los medicamentos GLP-1 atraviesan la barrera hematoenceflica y reducen la sealizacin de dopamina en el principal centro de recompensa del cerebro, haciendo que las sustancias adictivas resulten menos gratificantes

Similarly, reduction of VSMC Glp1r expression did not perturb body weight, glucose tolerance, or the glycemic response to semaglutide (Figure 1, OQ)

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