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Also, mice carrying a genetic mutation that lack one of the igf1r alleles ( igf1r +/ ) show a 10 % reduction in post-natal growth, insulin resistance and glucose intolerance [237]

In this pathway, copper-induced oxidative stress is thought to arise mainly from interference with mitochondrial enzymes rather than from copper directly generating ROS [23]

Similarly, the expression of TGFB1 and its downstream target CTGF , two key pro-fibrotic factors were not affected by Exendin-4, suggesting that GLP-1 does not induce a pro-fibrotic phenotype, though a change in the mechanical property by changing the ratio of collagen/elastin 3 cannot be excluded

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