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The role of GIP in response to DPP-4 inhibition To test the specific contribution made by GIP to linagliptin-induced improvements in glucose tolerance, a GIP antagonising antibody (GIPrAB) [10] was administered to littermate wild-type and Gcg RA Null mice

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Notably, NAC elevated striatal levels of glycogen synthase kinase 3 (GSK3), a key regulator of dendritic structure, which led to significant recovery of dendritic complexity and spine density

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