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In the rat model, bidirectional effects on nitric oxide synthases have been observed: stimulation of the endothelial form (eNOS) with simultaneous inhibition of the inducible form (iNOS), suggesting a role in maintaining vascular homeostasis

This resistance of CRC cells to 5-FU can be linked to the activation of the PI3K/AKT signaling axis (Azwar et al

Development of co-infectious models of AD in non-human primates would help clarify findings from clinical samples, although such investigations would only demonstrate the causality of microbes in MAD to the extent of the microbes tested

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