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Subsequently, the tumor tissue was pathologically analyzed

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Abnormal lipid metabolism, including dyslipidemia, is a significant risk factor for cardiovascular diseases

[22] The same work parsed the downstream genes carefully and found a split: some responses (such as induction of the survival gene Bcl-XL) were abolished in mice engineered to lack functional IGF-1 receptors on their muscle fibers, while others (such as induction of matrix metalloproteinase-13) persisted even without the receptor