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(33) documented that acute gastric lesions in rats, induced by ischemia followed by reperfusion (I/R) can be attenuated by antioxidant enzymes, such as SOD, catalase and by NO-synthase inhibitors, suggesting that ROS may cooperate with NO in I/R-induced gastric damage and that reduction in ROS and NO toxicity is required to achieve satisfactory protection against gastric lesions induced by I/R

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