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One possible explanation for the similar effects on weight loss observed with both GIPR agonism and antagonism might be that chronic pharmacological GIPR activation induces functional antagonism in a subset of neurons through receptor desensitization, as seen in adipocytes, isolated pancreatic islets and mouse neuroblastoma Neuro2a cells 36,37

The sequence contains norleucine, aromatic residues, and basic side chains, giving the compound a mixed hydrophobic and cationic profile in laboratory characterization

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