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The aim of this review was to summarize folic acid, vitamin B12 and homocysteine metabolism in CKD patients and to analyze the published evidences on folic acid and vitamin B12 deficiency as cardiovascular risk markers and therapeutic targets in CKD and ESRD patients

reported that downregulated IIS increased tau phosphorylation, promoted GSK activation, and decreased insulin receptor substrate-1 (IRS1), phospho-Akt, drebrin, and postsynaptic density (PSD95) resulting in cognitive impairment in the HFD model [156]