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Additional proposed mechanisms include improved myocardial energetics, reduced inflammation, and attenuation of cardiac remodeling, although the relative contribution of each pathway remains uncertain [33,34,35]

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The activation of PLC results in the release of inositol-1,4,5-trisphosphate (IP 3 ) from membrane associated phosphatidylinositol-4,5-bisphosphate (PIP 2 ), which then binds to specific IP 3 receptors in the endoplasmic reticulum/sarcoplasmic reticulum (ER/SR) resulting in the release of intracellular stores of Ca 2+

doi:10.1016/0165-1781(89)90047-4