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In a 2001 study published in Endocrinology , Heffernan and colleagues reported that chronic AOD-9604 administration reduced body weight gain, decreased adipose tissue mass, and increased fat oxidation in obese mice without altering IGF-1 levels or glucose metabolism effects that were abolished in beta3 adrenergic receptor (3-AR) knockout mice, implicating 3-AR signaling as the mechanism of action [1]

HDACs and associated epigenetic functions have been suggested as potential therapeutic targets for PD (Sharma and Taliyan, 2015a

A 2021 study published in Clinical Infectious Diseases found that Ta1 treatment in severe COVID-19 patients significantly reduced mortality and improved immune recovery

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