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Upon the prevention of IGF-IR ligand-binding and activation by anti-IGF-IR mAb, InsR may replace IGF-IR to bind IGF2 and insulin, leading to a compensatory increase in InsR phosphorylation/activation and sustained IGF/insulin signaling

[DOI] [PubMed] [Google Scholar] 50.Moran C.N., Pitsiladis Y.P