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Target mRNA abundance was normalized to a factor derived from the geometric mean of expression values for -actin and Gapdh mRNAs calculated using the GeNorm program (Pabona et al., 2012)

Its been studied for its ability to: Support tendon and ligament repair Improve angiogenesis (formation of new blood vessels) Reduce inflammation in the gut and liver Possibly influence neurotransmitter systems like serotonin and dopamine While most studies are in animals, these pathways overlap with some of the most challenging aspects of menopause

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