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Clopidogrel is inactive when administered It must be metabolized in the liver (mainly by CYP2C19) into an active thiol metabolite The active metabolite then produces the antiplatelet effect Mechanism of action The active metabolite irreversibly inhibits the P2Y ADP receptor on platelets This prevents platelet activation and aggregation Clinical uses Prevention of ischemic stroke Prevention of myocardial infarction Postcoronary stent placement Alternative to aspirin in aspirin intolerance Delayed onset compared to aspirin (because activation is required) Reduced efficacy in patients with CYP2C19 deficiency or when taken with CYP2C19 inhibitors (e.g., omeprazole) Effect lasts for the life of the platelet (710 days) Clopidogrel = Prodrug Aspirin = Active drug Prasugrel, Ticagrelor antiplatelets (note: ticagrelor is NOT a prodrug) To view or add a comment, sign in With respect to gliptins and Parkinson's disease, which statement is false

It has a key role in cellular protection, detoxification processes and strengthening the immune system

The company's GLP-1 page states that licensed clinicians "follow strict medical protocols to protect patient safety and apply clear exclusion criteria to determine who may or may not qualify for GLP-1 treatment." Partner pharmacies fulfill prescriptions written by the medical providers

Now we will examine the growing evidence of anti-addictive properties and mental health benefits of GLP-1s