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[26] DNA methylation is involved in the functioning of genes and is an essential epigenetic control mechanism in mammals

They: Produce collagen Organize extracellular matrix Restore tensile structure In experimental tendon transection models, BPC-157 has been associated with: Increased fibroblast density Enhanced migration into injury site Accelerated granulation tissue formation Proposed signalling involvement includes: FAK (Focal Adhesion Kinase) activation ERK1/2 signalling Src pathway modulation FAK signalling is particularly important because it regulates: Cell adhesion Cytoskeletal reorganization Migration along collagen scaffolds Tendon strength depends not just on collagen quantity but alignment

All this requires continuous monitoring and thoughtful planning to minimize risks, especially with the possibility of metabolic changes and side effects

doi:10.1186/s12903-016-0215-y