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Pre-clinical models confirm it: rats dosed with liposomal GSH showed 40% less paw oedema and lower rheumatoid factor levels, mirroring the potential in humans

In addition, our in vitro studies indicate a novel mechanism, that is GSTP can be phosphorylated at Ser184 and enter the nucleus under LPS stimulation, and GSTP interacted with HMGB1 to inhibit cPKC-mediated phosphorylation of HMGB1 and association between HMGB1 and CRM1 in the nucleus

Comparing differential gene expression between primary and induced hepatocytes may identify targets to enhance cell competence for transplantation