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doi: 10.14202/vetworld.2023.1541-1545 25 RehmanMSRehmanSUYousafWHassanFUAhmadWLiuQet al

Patients with certain genetic profiles show stronger blood pressure improvements, allowing PlexusDx to personalize your therapy and set realistic expectations for your cardiovascular outcomes

This is especially true during the early period after starting semaglutide and during dose escalation phases

Research has identified multiple pathways through which semaglutide impairs normal stomach function: Direct inhibition of gastric motility through vagus nerve modulation Disruption of gastric pacemaker cells that coordinate stomach contractions Impaired gastric accommodation in the fundus and body regions Antral hypomotility preventing proper food grinding and propulsion Dysregulation of pyloric sphincter relaxation blocking stomach emptying Alterations in small intestinal motility affecting the entire digestive process Clinical data reveals that up to 75% of patients with normal gastric emptying before receiving GLP-1 agonists developed delayed gastric emptying after treatment initiation, with some studies showing 30% experiencing severe delays qualifying as gastroparesis