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Helicobacter pylori cholesterol glucosylation modulates autophagy for increasing intracellular survival in macrophages

To further confirm that the p62-Keap1-NRF2 axis is responsible for the high expression of GPX4 in EBV-infected cells, we examined the effect of NRF2 on GPX4 expression in EBV-negative and EBV-positive NPC cells using siRNAs against NRF2