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Treating cells with L-buthionine sulfoximine (BSO), an inhibitor that blocks the synthesis of GSH, renders neuronal SH-SY5Y cells expressing mutant valosin-containing protein (VCP) R487H more susceptible to oxidative stress (Hirano et al., 2015) and increases mutant SOD1 toxicity (Alvarez-Zaldiernas et al., 2016

As with all findings in this category, results are early-stage and not a basis for medical claims

The importance of dietary protein cannot be underestimated in this population since inadequate protein intake contributes, among other things, to a decrease in lung reserve capacity, increased skin fragility, osteoporosis, decreased immune function and muscle mass (sarcopenia), poor healing and longer recuperation from illness [2, 47]

The Genetic Switch: Understanding Nrf2 Activation If glutathione is conceptualized as the primary fuel of the cellular antioxidant system, a crucial question concerns the regulatory engine that governs its synthesis and utilization