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Keap1 responds to oxidative stress signals or chemical inducers by freeing Nrf2

It provides the cysteine residue (the rate-limiting amino acid in glutathione synthesis) and supports endogenous GSH production

By further elucidating the pathogenic mechanism by which hepatic iron accumulation triggers ferroptosis through the c-Myc-Acsl4 regulatory axis, thereby accelerating the progression of metabolic-associated steatohepatitis (MASH), our research team identified a novel iron chelator, FOT1 (FerroTerminator1, formerly known as CN128), through drug screening, which can prevent and treat the onset and progression of MASH disease [117]

Lactate levels are a biomarker of tissue hypoxia as micro hemodynamic status