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Deficiency of GPx1 also promotes pro-inflammatory activation of endothelial and other cell types, and accelerates atherogenesis in susceptible animal models [6, 78, 79, 84, 85]

In the stimulated group, platelet aggregates rapidly formed on the endothelial surface, and continued to accumulate over time, while leukocytes adhered to the vessel walls and began migrating through the endothelium into the collagen matrix after 1 h of blood perfusion

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