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Safety data in humans is extremely limited because these peptides lack clinical trial evaluation for vascular applications

These effects are physiologically distinct from insulin (which drives cellular glucose uptake) and from GLP-1 agonists (which act on a different incretin receptor) the rationale is to replace the missing second beta-cell hormone rather than add more insulin

Part of what I try to do is not reduce histamine in the nervous system but reduce the histamine in the gut, which is perpetuating a lot of the gut inflammation

Anti-leukemia efficacy of the dual BCL2/BCL-XL inhibitor AZD0466 in acute lymphoblastic leukemia preclinical models