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Based on earlier data, there was a lot of expectation that CagriSema would come out on top

Ub is activated by E1, which hydrolyzes ATP and binds Ub to its cysteine residues, as the initial stage in the binding of Ub to substrate proteins

In line with this assumption, in vitro insulin responses to glucose from NKCC1-expressing islets but lacking NKCC2A were normal (Kelly et al., 2019)

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