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[1] [2] Low blood glucose can trigger compensatory tachycardia and palpitations

Kidney transplant recipients exhibit characteristic dysbiosis patterns paralleling those in T2DM, including depletion of SCFA-producing taxa (e.g., Roseburia , Faecalibacterium ) and reduced Akkermansia muciniphila abundance ( The following molecular mechanisms are implicated: SCFA (butyrate) depletioncompromised intestinal tight junction integrity endotoxemia (LPS translocation)TLR4/NF-B-driven systemic metabolic inflammation and insulin resistance