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Since this characteristic strongly correlates with tumorigenicity in animals [25], we hypothesized that interventions inhibiting TIC growth in 3D gels could also impede tumor development in vivo

To this end, we evaluated multiple cellular parameters including loss of cell viability, caspase 3 activation, GSH-conjugate formation, GSH homeostasis, and cellular stress response pathway activation in mock- and GSTP1 -transfected HepG2 cells

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153 Overall, in contrast to earlier observations, Ang A is not a partial agonist with greater affinity for AT 2 receptors than Ang II, but displays similar in vitro and in vivo properties as Ang II