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Treatment of early ring-stage artemisinin resistant (Cam3.II R539T ) and sensitive (Cam3.II REV ) parasites with 10 mM of BSO for 1 h significantly depleted both -Glu-Cys and reduced GSH levels in both lines compared to untreated controls (Fig 5A)

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The molecule was developed by Frank Ng's group at Monash University and Metabolic Pharmaceuticals in Australia during the 1990s and 2000s as a potential anti-obesity drug intended to isolate the lipolytic region of hGH without activating the growth hormone receptor or raising IGF-1.nnIn published preclinical work, the fragment has been shown to stimulate lipolysis, suppress lipogenesis, and modulate the beta-3 adrenergic receptor pathway in adipose tissue rather than acting through the classical hGH receptor