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We propose that curcumin exerts its broad therapeutic effects via a unified inflammationoxidative stress axis, primarily by inhibiting NF-B and NLRP3 activation, reducing ROS and pro-inflammatory cytokines (TNF-, IL-1/18), as seen across models of depression, metabolic disorders, and neurodegeneration
There can also be fluctuations between the two conditions
Variations in genes such as ACTN3, which encodes alpha-actinin-3, a protein critical for fast-twitch muscle fibers, can affect muscle performance and fatigue resistance
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