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Description

The selectivity profile confirmed in multiple rodent and early human studies allows researchers to attribute observed experimental outcomes specifically to GH-axis modulation rather than to concurrent adrenal or lactotroph activation, a significant methodological advantage over the hexapeptide GHRPs

Hypofunction of NMDARs, particularly in inhibitory parvalbumin (PV) interneurons (357), may initiate the disease process, leading to decreased inhibition of pyramidal neurons

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