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As neutrophils begin their transepithelial migration into the lungs, they become primed to phagocytose invading microbes and release toxic mediators including reactive oxygen species (ROS), neutrophil elastase, proteases, and nitric oxide (NO)

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In ALS-mouse models, non-functional proteins secondary to the oxidation of amino acid residues by peroxynitrite have been observed (Yoshino and Kimura, 2006)