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Pathogenetic role of eNOS uncoupling in cardiopulmonary disorders

Since podocyte injury extends beyond Gb3 accumulation, analysis of signaling networks in Fabry have shown the involvement of -synuclein in lysosomal impairment and hints to pharmacological treatments modulating -synuclein and the use of 2 -adrenoreceptor agonists [65] as promising interventions

HepG2 cells were obtained from European Collection of Cell Cultures (ECACC, Salisbury, United Kingdom)

Many high-performing professionals feel more comfortable discussing health vulnerabilities when theyre in their own space, without the clinical environment triggering defensiveness