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The study also documented increased skin density and thickness, indicating structural improvements beyond surface-level changes

About this article Cite this article Aguirre, G.A., De Ita, J.R., de la Garza, R.G

Mechanistically, oxidative stress triggers endoplasmic reticulum stress through the activation of the PERK-eIF2-ATF4 and IRE1-XBP1 signaling pathways, as well as through the upregulation of CHOP (C/EBP homologous protein), which promotes apoptotic signaling (75) as shown in Figure 4