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Targeting zinc or zinc transporters for tumor therapy shows promise, but potential toxic effects must be considered

The cellular and molecular origin of reactive oxygen species generation during myocardial ischemia and reperfusion

Another report demonstrated the role of ferroptosis in the pathogenesis of mitochondrial disease-associated epilepsies and found that -tocotrienol quinone (EPI-743) potently dose-dependently ameliorated ferroptosis in the cells of patient of five distinct pediatric epilepsy syndromes by reducing LPO and 15-hydroxyeicosatetraenoic acid (15-HETE), a specific 15-LO product ( 3 )-induced posttraumatic epilepsy mouse model and in ferric ammonium citrate (FAC)-induced HT22 hippocampal neuron cells by decreasing lipid ROS and 4-HNE, downregulating prostaglandin endoperoxide synthase 2 (PTGS2), and inhibiting the expression of 12/15-lipoxygenase (12/15-LOX)