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In conclusion, this study systematically reveals for the first time the molecular mechanism by which PLB improves ovarian function in PCOS mice by inhibiting the YTHDF1/SLC7A5 axis, modulating m6A methylation modification, and inhibiting the ferroptosis pathway

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The BTBR mice show an overall reduction in tyrosine hydroxylase (TH), a key enzyme in dopamine synthesis, while the Fmr1 -KO mice show abnormal TH-positive axons morphology [60]

PNPO mutations are treated with high doses of P5P (active form), thus providing the active form of B6 without the need for conversion