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It has been hypothesized that GHK-Cu may alter the expression of a wide array of genes associated with cellular repair, inflammation signaling pathways, and oxidative stress responses
Concurrently, lack of Nrf2 decreased protein aggregation and cardiac hypertrophy and improved cardiac contractility in the mice transgenic for the mutant human B-crystallin, suggesting that the sustained upregulation of Nrf2 and subsequent increase in GSH-mediated removal of hydrogen peroxide contributes to the reductive stress phenotype [51]
Growth factor modulation: It enhances the activity of growth hormone receptors, fibroblast growth factor, and EGF, all critical to tissue repair
Many polyphenols, including curcumin and quercetin, undergo rapid metabolism, reducing their therapeutic efficacy
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