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In this context, the present review aimed to summarize current knowledge on the biological role of carnitines as modulators of mitochondrial metabolic activity, to provide the basis for targeted in vitro and in vivo use of carnitines to sustain and restore oocyte competence and female fertility 2

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consistent with the findings of a similar study, our results revealed that PFOS induced a concentration-dependent reduction in mitochondrial bioenergenesis, demonstrating that the administration of PFOA and PFOS (50200 M) induced the production of ROS, dissipation of mitochondria membrane potential, and apoptosis of HepG2 cells 31