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& Wang, B

doi: 10.1016/j.chom.2020.09.004 139 WuS.LiY.XuY.LiQ.ChuY.HuangR.et al

These are descriptions of published laboratory investigation, not statements about what the compounds do in people

Interestingly, another mechanism was proposed as an alternative way to stabilize NRF2 through the interaction between the autophagy adaptor protein p62/sequestosome-1 (p62/SQSTM1) and KEAP1, which leads to the sequestration of KEAP1 in the autophagy-deficient cells and enables NRF2 accumulation [20]