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It does not directly stimulate fibroblast proliferation under normal culture conditions, but substantially increases fibroblast survival under hydrogen peroxide-induced oxidative stress acting as a cytoprotective enhancer of the endogenous repair response rather than a direct mitogenic agent
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Several factors, such as diet, microbial metabolism of tryptophan, and endogenous enzymatic activity, can provide critical signals to the host, aiding in resisting colonization and defending against mucosal inflammation [51]
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