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Mechanistic work demonstrated that GLP-1 signalling can also modulate intestinal epithelial integrity, mucosal immune responses and the microbiome in pre-clinical colitis models, providing a biologically plausible rationale for a role beyond weight reduction [2]

Given the known link between iron deficiency and RLS, this may represent a key pathophysiologic pathway contributing towards the elevated RLS risk observed in our study

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Lower doses of LA are even adverse for hepatic fat metabolism in normal diet with 4% fat content