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Cell type- and etiology-specific regulatory blank spots: Most research centers on renal TECs, but the role of non-parenchymal renal cells in ferroptosis-mediated AKI-to-CKD transition is largely understudied

Our literature review [25,26,27] in combination with updated search identified approximately 80 published MRS studies on ASD

Dephosphorylation of GlcNAc-Ins-P by the phosphatase MshA2 generates GlcNAc-Ins which is the substrate for the deacetylase MshB

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