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Elevations in a set of short-chain acyl-carnitines (C3 and C4) in posttreatment subjects speculates the dysfunction of CPT1 and CAT

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Moreover, high-dose L-carnitine supplementation completely prevented the reduction in mitochondrial function markers (Figures 5C, D), and ultimately attenuated cytotoxicity (Figure 5E) in lenvatinib-treated C2C12 myocytes