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Preclinical models demonstrate that region-specific elevations of central KYNA highlight a functional trade-off: while KYNA is often considered neuroprotective due to its antagonism of glutamatergic receptors, its broad receptor profile also allows it to inhibit 7nAChRs, which are essential for prefrontal cortical processing and mood regulation

367 The administration of liraglutide also impeded osteoclastic bone formation, thereby inhibiting bone resorption and exerting protective effects on bone health in T1D mice