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These lipid-laden cells demonstrate increased expression of astrogliosis markers (e.g., GFAP) at mRNA and/or protein levels, promoting ER stress-mediated release of proinflammatory cytokines that induce reactive astrogliosis and microglial activation (98, 99)

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Circulating glucose, insulin, and leptin tune hypothalamic circuits that gate hunger and reward

Graphical Abstract The increasing use of glucagon-like peptide-1 receptor agonists (GLP-1 RAs) raises concern about potential interactions with narrow therapeutic index medications such as warfarin