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The mechanism isn't fully mapped, but it likely involves circadian modulation of vagal afferent sensitivity

In the combined absence of IL-25 and IL-33 signalling, nuocytes fail to expand, resulting in a severe defect in worm expulsion that is rescued by the adoptive transfer of in vitro-cultured wild-type, but not IL-13-deficient, nuocytes

and people with an MTHFR genetic polymorphism (or mutation) whose cells are less efficient at converting folate into its active form