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Preclinical models demonstrate that region-specific elevations of central KYNA highlight a functional trade-off: while KYNA is often considered neuroprotective due to its antagonism of glutamatergic receptors, its broad receptor profile also allows it to inhibit 7nAChRs, which are essential for prefrontal cortical processing and mood regulation

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Luminal l-valine (50 mM) strongly stimulated GLP-1 release from the perfused rat small intestine ( P 0.05), suggesting that co-transport of l-valine and Na + is not important for the depolarization necessary to activate the voltage-gated Ca 2+ -channels