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As expected, most of the fluorescence signals in the three groups were mainly observed in the dermis but not in the epidermis layer

Moreover, classical methylation changes in lung cancersuch as silencing of tumor suppressors p16 and RASSF1Afurther illustrate the importance of aberrant DNA methylation in tumorigenesis [71,72,73,74,75].Together, these observations support the notion that shared methylation-regulated pathways, exemplified by KCNK3, may underlie the molecular crosstalk between hypertension and lung cancer

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