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Studies utilizing models of myocardial infarction have demonstrated reduced infarct size, improved cardiac function, and enhanced neovascularization following TB-500 treatment
Indeed, Sp was able to restore cytosolic and mitochondrial redox balance in neuronal cell line SHSY-5Y subjected to 1-methyl-4-phenylpyridinium (MPP + ) treatment which is known to cause mitochondria dysfunction and oxidative stress through complex I inhibition 1 (Fig
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