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We assessed Np63 expression in LUSC cell lines, selecting one cell line with low (NCI-H520) and two cell lines with elevated (NCI-H1703 and HCC95) endogenous levels of Np63 expression (Fig

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Target sequence was selected by CRISPR-PLANT ( All the vectors were transformed into Agrobacterium tumefaciens strain GV3101 using electroporation

Redox imbalance is a key pathogenic mechanism in melanoma and non-melanoma skin cancers, with activation of the NRF2-ARE pathway acting as an intrinsic defense against oxidative stress