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Targeting Downstream Effectors of ER Activation While ER-targeted therapies are likely to suffer from some of the same risks as estrogen supplementation (115), targeting selected downstream effectors of ER may represent an opportunity to capture the metabolic benefits while bypassing adverse effects associated with estrogens

RNA sequencing suggested enhanced negative regulation of ferroptosis in the GSH-treated grafts, which showed reduced lipid peroxides, better mitochondrial ultrastructure, and SLC7A11/GPX4 axis activation

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The specific neurons targeted differ