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For example, in alveolar macrophages, GLP-1 receptor agonism suppressed activation of the NLRP3 inflammasome and reduced production of IL-1 [65], an effect in part mediated by the disruption of histone lactylation, a recognised regulator of pro-fibrotic gene expression [65]

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Research suggests that IGF1 LR3 may preferentially start certain isoforms of the IGF-1 receptor or may have differential effects on many tissue types due to differences in receptor density, co-receptor expression, or downstream signaling components